Reactions

Reversible reactions are represented as bidirectional arrows (e.g., reactions 1 and 2 in the EGFR scheme below) and irreversible reactions as unidirectional arrows (Reaction 4 in EGFR). Enzyme reactions are drawn as an arrow with two bends, where the enzyme is located on the middle segment (Reaction 3 in EGFR). Numbers for identifying reactions are located on the forward (kf) side of the bidirectional arrows. For clarity, the same reactant may be represented multiple times in a given reaction scheme, but it is modeled only as a single reactant.

Reaction A: EGFR pathway, SoS

A1 A2 A3 A4 A5 A6 A7 A8 A9 A10 EGFR SHC Grb2 SoS


Reaction B: Ras

B1 B2 B3 B4 B5 B6 B7 B8 B9 B10 B11 B12 B12 B12 B13 B13 B13 inact_GEF GAP GDP_Ras

Reaction C: AC, PDE, cAMP

C1 C2 C3 C4 C5 C6 C7 C8 C9 C10 C11 C12 C13 C14 C15 C16 C17 C18 ATP ATP AC1 AC2 cAMP_PDE PDE1

Reaction D: Glu, mGluR, Gq

D1 D2 D3 D4 D5 D6 D7 D8 D9 G_GDP mGluR


Reaction E: AA, PLA2

E1 E2 E3 E4 E5 E6 E7 E8 E9 E10 E11 E12 E13 E13 E13 E13 E13 PIP2* PIP2* PLA2_cyt APC APC APC APC APC

Reactions F and G: PLC_beta, PLC_gamma, DAG, IP3

F1 F2 F3 F4 F5 F6 PLC_g PIP2 PIP2 G1 G2 G3 G4 G5 G6 G7 G8 G9 PIP2 PIP2 PLC

Reaction H: MAPK Cascade

H1 H2 H3 H4 H5 H6 H7 H8 H9 H10 H11 H12 H13 MKP1 MKP1 PP2A PP2A PP2A PP2A craf_1 MAPKK MAPK


Reaction I: CaMKII

The CaM-Kinase model takes into account the fact that the enzyme exists as an 8 to 12-mer. We compute all autophosphorylation rates as intra-holoenzyme but inter-subunit rates by treating autophosphorylations as zeroth order reactions, i.e., no concentration terms. The various phosphorylation states of CaMKII have different enzyme kinetics, and each of these were explicitly modeled. For simplicity the autophosphorylation steps are represented by a single enzyme arrow in this figure, with CaMKII_a as the combined activity of the various phosphorylation states. The individual kinetic terms used in the model are indicated by the multiple rate references on the arrows. I1 I2 I3 I4 I5 I6 I7 I8 I9 I10 I11 I12 I13 I14 I15 I16 CaMKII

Reaction J: PKA

J1 J2 J3 J4 J5 J6 J7 R2C2 PKA_inhibitor

Reaction K: PKC

K1 K2 K3 K4 K5 K6 K7 K8 K9 K10 PKC_inactive


Reaction L: Ca, IP3

L1 L2 L3 L4 L5 L6 L7 L8 L9 Ca Ca_stores Ca_stores Stores_Leak Ca_transp IP3R Ca_pump Ca_ext Extracell_Leak Cap_channel Ca

Reaction M: CaM

M1 M2 M3 M4 M5 M6 M7 M8 CaM neurogranin


Reaction N: CaN

N1 N2 N3 N4 N5 Calcineurin

Reaction O: PP1

O1 O2 O3 O4 O5 O6 O7 O8 O9 PP1_active PP2A I1 I1 I1

NMDA Receptor

The NMDA Receptor was modeled according to De Schutter and Bower 1993 (Reference xxyy) in a compartmental model of a hippocampal CA1 neuron which was implemented in GENESIS. The reaction scheme is as follows:

A + R <---> AR
Kr = 50 uM

AR ---> AR*
tau_g = 283 sec

AR* ---> A + R
tau_d = 0.002 msec

A ---> removal
tau_h = 5.88 msec.


AMPA Receptor

The NMDA Receptor was also modeled according to De Schutter and Bower 1993 (Reference xxyy) in a compartmental model of a hippocampal CA1 neuron which was implemented in GENESIS. The same reaction scheme was used as for the NMDA receptor:

A + R <---> AR
Kr = 2 uM

AR ---> AR*
tau_g = 600 msec

AR* ---> A + R
tau_d = 0.001 msec

A ---> removal
tau_h = 1.25 msec.


Depolarization

The depolarization was computed using a compartmental model of a hippocampal CA1 neuron which was implemented in GENESIS. The Crank-Nicholson implicit integration method was used (Bhalla, Bilitch and Bower 1993, Hines 198xx xxyy refs).


Regulation of nuclear and cytoskeletal events

These events were not explicitly modeled. Instead, the production of MKP-1 (which is produced following MAPK induction) was modeled by raising MKP-1 concentration to various levels for various durations following tetanic stimulus. See Figure 2 G and H of the original paper.


Main page